1、 Samanta A*, Li B*, Song X, Bembas K, Zhang G, Katsumata M, Shen Y, Saouaf SJ, Hancock WW, Wang Q and Greene MI. (2008) TGF-beta and IL-6 signals modulate chromatin binding and promoter occupancy by acetylated FOXP3. THE PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES. 105(37):14023-7. Epub 2008 Sep 8 (*co-first authors) 2、 Zhou Z, Song X, Li B*, and Greene MI*. (2008) FOXP3 and its partners: structural and biochemical insights into the regulation of FOXP3 activity. IMMUNOLOGIC RESEARCH. Epub 2008 July 15th DOI. 10.1007/s12026-008-8029-x. Review. (*co-corresponding authors). 3、 Zhou Z, Song X, Berezov A, Zhang G, Li Y, Zhang H, Murali R, Li B*, and Greene MI*. (2008) Human Glucocorticoid Induced TNF Receptor Ligand Regulates Its Signaling Activity Through Multiple Oligomerization States. THE PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES. 105(14):5465-70. (*B.L. and M.I.G designed the research). 4、 Li B and Greene MI. (2008) FOXP3 Biochemistry in regulatory T cells. How diverse signals regulate suppression. IMMUNOLOGY. 123 (1), 17–19. The 50th anniversary issue, Feature in Tsup/Teff cells. Review. 5、 Li B, Samanta A, Song X, Iacono KT, Tao R, Shen Y, Riley JL, Hancock WW, Saouaf SJ, Greene MI. (2007) FOXP3 interactions with histone acetyltransferase and class II histone deacetylases are required for repression THE PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES. 104 (11): 4571-4576 (Research Highlighted in Nature Immunology May 2007; 8 (5): 471) 6、 Li B, Samanta A, Song X, Iacono KT, Brennan P, Chatila TA, Roncador G, Banham AH, Riley JL, Wang Q, Shen Y, Saouaf SJ, Greene MI. (2007) FOXP3 is a homo-oligomer and a component of a supramolecular regulatory complex disabled in the human XLAAD/IPEX autoimmune syndrome. INTERNATIONAL IMMUNOLOGY 19 (7): 825–835. 7、 Li B and Greene MI. (2007) FOXP3 actively represses transcription by recruiting the HAT/HDAC complex. CELL CYCLE June 15, 2007; 6 (12): 1432-1436. Perspective. 8、 Li B, Saouaf SJ, Samanta A, Shen Y, Hancock WW and Greene MI. (2007) Biochemistry and therapeutic implications of understanding mechanisms underlying FOXP3 activity. CURRENT OPINION IN IMMUNOLOGY. 19(5):583-8. Epub 2007 Aug 20. Review. 9、 Li B, Samanta A, Song X, Furuuchi K, Iacono KT, Kennedy S, Katsumata M, Saouaf SJ, Greene MI. (2006) FOXP3 ensembles in T cell regulation. IMMUNOLOGICAL REVIEW. 2006 Aug; 212:99-113. Review. 10、 Li B, Song X, Samanta A, Bembas K, Brown A, Zhang G, Katsumata M, Shen Y, Saouaf SJ and Greene MI. (2008) FOXP3 biochemistry will lead to novel drug approaches for vaccines and diseases which lack suppressor T cells. The Immune Synapse as a Novel Target for Therapy, p147-154.? The series PROGRESS IN INFLAMMATION RESEACH. Book chapter. 11、 Li B*, Shen Y, Zhou Z, Song X, Bembas K, Zhao XY, Cai Z, Berezov A, Saouaf SJ, Zhang HT, Wang Q and Greene MI*. (2008) Epigenetic regulation of autoimmune diseases through deacetylase inhibition. THE EPIGENETICS OF AUTOIMMUNE DISEASE, edited by Prof. Moncef ZOUALI (Paris, France), John Wiley & Sons, Inc. Book chapter. In press. (*co-corresponding authors). 12、 Tao R, de Zoeten EF, Ozkaynak E, Chen C, Wang L, Porrett PM, Li B*, Turka LA, Olson EN*, Greene MI*, Wells AD, Hancock WW, (2007) Deacetylase inhibition promotes the generation and function of regulatory T cells. NATURE MEDICINE, 13(11):1299-307 (*B.L., M.I.G and E.N.O. were instrumental in early stages of the project; Research Highlighted in Science 26 October 2007:?Vol. 318. no. 5850, p. 537; also in Nature Reviews Drug Discovery 6, 958 December 2007). 13、 Tao R, de Zoeten EF, Ozkaynak E, Wang LQ, Li B, Greene MI, Wells AD and Hancock WW, (2007) Histone deacetylase inhibitors and transplantation. CURRENT OPINION IN IMMUNOLOGY. 19(5):589-595. Review. 14、 Brennan PJ, Saouaf SJ, Van Dyken S, Marth JD, Li B, Bhandoola A, Greene MI. (2006) Sialylation regulates peripheral tolerance in CD4+ T cells. INTERNATIONAL IMMUNOLOGY 18(5):627-35. 15、 Hirohashi Y, Wang Q, Liu Q, Li B, Du X, Zhang H, Furuuchi K, Masuda K, Sato N, Greene MI, (2006) Centrosomal proteins Nde1 and Su48 form a complex regulated by phosphorylation. ONCOGENE 25(45):6048-55.
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